ever see game-time action for the Hogs? Gut feelings?
I’m skeptical, but the last time he was asked about it, Anderson sounded positive. From Bob Holt’s article in December:
Anderson said he’s “very optimistic” Garland will be able to play next season. He has been practicing on a limited basis, but isn’t allowed to scrimmage or have contact.
“He’s been good,” Anderson said. “I think the biggest relief came to him when he could actually get out on the floor and do some things with us.
“Not necessarily the scrimmaging part of it, but just to be able to get out there. As long as he’s with these guys, he’s OK.”
I’m disappointed Garland hasn’t been cleared to play but it’s a blessing that the doctors found the medical issue and he getting treatment. I pray he gets cleared to play but the young man’s health and welfare is the most important thing to worry about not basketball!
Question for any doctor’s on this forum as to what heart condition could he possibly have that was not detected until he arrived at the University and what treatment options are there associated with the condition and success rates of treatments?
All I have read or heard is that he has a heart condition. How was it detected, EKG or other tests?
I know everyone is hopeful and I hope it works out for him.
But right now there has been nothing to change his situation.
They all have a very extensive physical once they get to campus.
His issue was detected during the physical and then further tests were done to validate the initial finding.
haven’t heard enough details & won’t speculate on what the kid has
no one can make more than a guess, but the caution exhibited and lack of prior symptomatology strongly suggests HOCM which is hypertrophic cardiac myompathy. Real formal definition from ACC:
Easier read although still thorough is Cleveland Clinic blurb:
Definition and Etiology
septal hypertrophy of the left ventricle
Figure 1: Click to Enlarge
Hypertrophic cardiomyopathy (HCM) is defined as hypertrophy of the myocardium more than 1.5 cm, without an identifiable cause (Figure 1). Other causes of left ventricular (LV) hypertrophy, such as long-standing hypertension, amyloidosis, and aortic stenosis must first be excluded before HCM can be diagnosed. As our understanding of the genetics of HCM continues to progress, the diagnosis of HCM will continue to incorporate information obtained from genetic testing, while also continuing to rely on transthoracic echocardiography (TTE) for the assessment of the phenotypic manifestations and the overall clinical severity of the disease.
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Prevalence and Risk Factors
HCM is the most common genetic cardiovascular disease.1 The estimated prevalence in the general adult population with phenotypic evidence of HCM is 1 per 500.2 Men are more often affected than women and black patients more so than white. In young adults, HCM is the most common cause of sudden cardiac death (SCD).3,4
Any strenuous exercise that increases afterload (i.e., heavy weight lifting/training) can theoretically increase the magnitude of LV hypertrophy over time and thus worsen obstruction in subjects with pre-existing HCM. Risk factors for the development of end-stage HCM (manifesting as LV systolic dysfunction and LV dilation) include younger age of onset/presentation of HCM, a family history of HCM, increased ventricular wall thickness, along with the presence of certain genetic mutations in certain individuals.
Thanks, Ray. I did read it. But I still don’t understand all of it. Guess that’s normal when you start getting through that.
My oldest daughter Sarah was flagged in a physical ahead of her junior year in college. They heard something that suggested a more detailed exam by a cardiologist was needed. She had a murmur. They figured it out. And, they let her play.
The funny thing when she came home from college to get further tests, they were stunned at her low heart rate. They asked if she ran marathons. No. They were worried. Then, the specialists laughed when it finally popped out that she was a college soccer player. The doc said, “Well, that’s the same thing.”
just a comparison to Nick Fairley, who is under the same setting:
A really good friend of mine is the chief medical officer for the NFL, no chances ever taken is what Allan said. He is a neurosurgeon, but knows his stuff. Len Bias and Hank Gaithers will not be duplicated. The only really dangerous condition is HOCM, if it were a dysrhythmia then I think there would be no secrecy. The secrecy is worrisome:
A long time ago I was a distance runner at OBU. A cross country race back then was 5 miles. We ran a “casual” 5 miles every morning at a 6 minute per mile pace, then had our more demanding workout at 4:00 in the afternoon. (Yes, I’m kind of bragging. I can’t believe I could actually do this.) The point is, we were in incredible shape. I remember having discussions with a friend on the soccer team about who was in better shape. We both concluded that neither of us could step on to the other’s playing field and do it justice. I could run seemingly forever at a good pace. Soccer athletes run, stop, sprint, run backwards and sideways, jog, walk, and jump for 40 minutes. And that’s just the first half.
Hank Gathers is a good point of reference here; hypertrophic cardiomyopathy is what killed him. Len Bias isn’t. Bias died of a cocaine overdose. Another Celtic, Reggie Lewis, did die of HOCM during an offseason workout.
Hope the kid gets to
Play but from reading this I’m doubtful. My question is this. How long do we give his condition before medical hardship is declared. It would free up a scholarship if that was the case and would keep him on scholarship so he could get his education.
I could be wrong, but I believe it’s been said that he keeps his scholarship even if it becomes a medical scholarship. As for the timeline of when he will be ruled out, I believe (again I could be wrong) CMA is waiting to see if he can play this year. So, I don’t think his scholarship would be used for this coming year (18-19), but I believe it would be for the 19-20 season.
I am not yet joining the pessimist club yet on this. The fact that he is still on the team, sits on the bench for all games plus what I hear from the family friends (I know they are biased) plus Khalil’s attitude gives me hope. I don’t see yet that either Mike Anderson or Khalil have given up on this. I don’t think they are going through the motions, there must be a ray of hope somewhere in there.
On the other hand what worries me is that Dudley has inside information and he says he has no hope.
This is sad. In my opinion, before the season started, Khalil was the best in our freshman class. Of course Gafford far exceeded my expectations. Coach Flanigan has said Khalil is the best finisher he has coached. He was unstoppable in open court.
I know Hogville isn’t a great source of info, but Alan Garland (Khalil’s dad) has posted updates sporadically about this issue. They all seem optimistic. Now, and individual claiming to be Khalil’s cousin (Alan didn’t deny it) posted what the issue actually was. If this was Khalil’s cousin and that was indeed the diagnosis that was posted, I don’t understand the negativity around the situation. A NBA player has the same issues and had a procedure that corrected it. My wife and mom also had the same issues and both had the procedure and it fixed the issues. What was explained to me is medicine is prescribed to people who aren’t at high risk for heart attacks (Khalil was prescribed medicine), and only people at high risk for heart attack (my mom was 70) go straight to the procedure. Now, the procedure is expensive. So, that kind of brings up two questions in my mind:
Can the Garland’s afford the procedure, will insurance pay for it?
If not, is it illegal for a booster to pay, and if a booster does pay would it make Khalil ineligible?
Dunno. Nowadays, especially with the Trump administration doing its best to eliminate Obamacare, it’s a crapshoot what health insurance will or won’t cover, never mind high deductibles and all that.
Absolutely illegal. That would be an extra benefit; a life-saving extra benefit to be sure, but that would end his UA career more certainly than a bad ticker would. Nor could a Hog-friendly doctor or clinic give him a Hog discount.
The university can’t pay for a player on scholarship?
I’m not sure of the NCAA rules on medical care for an athlete. If he tore an ACL during practice, that’s one thing. This condition is not related to his participation in basketball other than that it prevents it.
Ok, I did some research. NCAA requires that athletes have health insurance. It does not mandate what is covered. Most schools cover their athletes but again coverage varies from school to school. When Tyrone Prothro of Bama shattered his leg in a game, requiring multiple surgeries, it didn’t cost him a dime. Bama and it’s insurer covered everything. But that’s an on-field injury. I’m much less confident that a heart irregularity would be covered by the school.
That is consistent with what I hear except the procedure part. This is the first I heard of that.
All this is speculation, but I will point out that hypertrophic cardiomyopathy and dysrhythmia are not exclusive. In fact a lethal dysrhythmia is one of the most common causes of death in HCM. The cardiomyopathy creates an irritable myocardium which can result in abnormal electrical impulses causing ventricular tachycardia or fibrillation